REDOX & ENERGY METABOLISM

Mitochondrial complex I null cancer models reveal unspecific antiproliferative effects of complex I inhibitors – Copie
Mitochondrial complex I null cancer models reveal unspecific antiproliferative effects of complex I inhibitors – Copie 1125 1625 Metabolism & Cancer
5- Ivana Kurelac, University of Bologna, Italy lire plus
Targeting the metabolic vulnerabilities of metastatic colorectal cancer
Targeting the metabolic vulnerabilities of metastatic colorectal cancer 1125 1625 Metabolism & Cancer
8- Josep Tarragó-Celada, Universitat de Barcelona, Spain lire plus
Targeting the fatty acid oxidation in combination with chemotherapy induces complete pancreatic cancer regression in vivo
Targeting the fatty acid oxidation in combination with chemotherapy induces complete pancreatic cancer regression in vivo 1125 1625 Metabolism & Cancer
7- Gabriela Reyes Castellanos, CRCM, Marseille, France lire plus
C2M2NF, A core model of central metabolism with Carbon, Nitrogen and Folate pathways: application to One-carbon metabolism in cancer cells
C2M2NF, A core model of central metabolism with Carbon, Nitrogen and Folate pathways: application to One-carbon metabolism in cancer cells 1125 1625 Metabolism & Cancer
1- Jean-Pierre Mazat, Université de Bordeaux & IBGC-CNRS, Bordeaux, France lire plus
Mechanisms of resistance to ferroptotic cell death : the impact of bioenergetic pathways
Mechanisms of resistance to ferroptotic cell death : the impact of bioenergetic pathways 1125 1625 Metabolism & Cancer
4- Célia Gotorbe, Centre scientifique de Monaco, Monaco lire plus
The IF1 mitochondrial inhibitory protein of ATP synthase modulates the permeability transition pore in a human cancer call model
The IF1 mitochondrial inhibitory protein of ATP synthase modulates the permeability transition pore in a human cancer call model 1236 1749 Metabolism & Cancer
9- Valentina Giorgio, University of Bologna, Italy lire plus
Loss of glutaredoxin-2 contributes to tumor oncocytic transformation via differential protein s-glutathionylation and hampering of oxidative phosphorylation
Loss of glutaredoxin-2 contributes to tumor oncocytic transformation via differential protein s-glutathionylation and hampering of oxidative phosphorylation 1125 1625 Metabolism & Cancer
3- Monica De Luise, University of Bologna, Italy lire plus
Pathogenic mitochondrial DNA mutations are associated with ROS signature and are prognostic markers in hepatocellular carcinoma
Pathogenic mitochondrial DNA mutations are associated with ROS signature and are prognostic markers in hepatocellular carcinoma 1125 1625 Metabolism & Cancer
6- Camelia Alexandra Coada, University of Bologna, Italy lire plus
Forced lipid catabolism entails higher proliferation rate of cervical cancer cells by ROS-dependent HIF1α activation
Forced lipid catabolism entails higher proliferation rate of cervical cancer cells by ROS-dependent HIF1α activation 2560 2560 Metabolism & Cancer
2- Serena Castelli, University of Rome “Tor Vergata”, Italy lire plus
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