Forced lipid catabolism entails higher proliferation rate of cervical cancer cells by ROS-dependent HIF1α activation

Forced lipid catabolism entails higher proliferation rate of cervical cancer cells by ROS-dependent HIF1α activation

Forced lipid catabolism entails higher proliferation rate of cervical cancer cells by ROS-dependent HIF1α activation 2560 2560 Metabolism & Cancer
Pathogenic mitochondrial DNA mutations are associated with ROS signature and are prognostic markers in hepatocellular carcinoma
The role of adipocyte G0/G1 Switch Gene 2 (G0S2) in the crosstalk between cancer cells and adipocytes
Mitochondrial complex I null cancer models reveal unspecific antiproliferative effects of complex I inhibitors – Copie
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